Anales de la RANM

57 A N A L E S R A N M R E V I S T A F U N D A D A E N 1 8 7 9 S U P L E M E N T O ASPECTOS CLÍNICOS Y MOLECULARES DE LAS AFASIAS PROGRESIVAS DEGENERATIVAS Juan J. Zarranz XV Curso de fundamentos moleculares de la Medicina An RANM · Año 2018 · 135(02) · Supl.01 · páginas 52 a 58 1. Broca PP. Sur le principe des localisations céré- brales. Bull. Société d’Anthropologie de Paris 1861;2: 190–204 2. Broca PP. Perte de la parole, ramollissement chronique et destruction partielle du lobe antérieur gauche du cerveau. Bull. Société d'Anthropologie de Paris 1861;2:235-238. 3. Broca PP. Remarques sur le siège de la faculté du langage articulé, suivies d'une observation d'aphémie (perte de la parole). Bull. Société Anatomique de Paris 1861;6:330-357. 4. Dronkers NF, Plaisant O, Iba-Zizen MT, Caba- nis E. Paul Broca`s historic cases : high resolu- tion MR imaging of the brains of Leborgne and Lelong. Brain 2007 ;130 :1432-1441 5. K. Wernicke. Der aphasische Symptomen com- plex. Eine psychologische Studie auf anatomi- scher Basis. Breslau, Cohn und Weigert, 1874. 6. Binder JR. Current Controversies on Wer- nicke's Area and its Role in Language. Curr Neurol Neurosci Rep. 2017 Aug;17:58 doi. org/10.1007/s11910-017-0764-8 7. Binder JR. The Wernicke area: modern evidence and reinterpretation. Neurology 2015;85:2170- 2175 8. Tremblay P, Dick AS. Broca and Wernicke are dead, or moving past the classic model of language neurobiology. Brain and Language 2016;162:60-71 9. Dronkers NF, Ivanova MV, Baldo JV. What do language disorders reveal about brain-lan- guage relationships? From classic models to networks approaches. J Int Neurpsychol Soc 2017;23:741-754 10. Fridriksson J, den Ouden D-B, Hillis AE, Hick- ok G, Rorden C et al. Anatomy of aphasia re- visited. Brain 2018;141:848-862 11. Pick A. Über die Beziehungen der senilen Hir- natrophie zur Aphasie. Prager Med Wochen- schr 1892;17:165–167. 12. Mesulam M-M. Slowly progressive aphasia without generalized dementia. Ann Neurol 1982;11:592–598. 13. Mesulam M-M, Rogalski E, Wieneke C, Hurley R, Geula C, Bigio E et al Primary progressive aphasia and the evolving neurology of the lan- guage network. Nat Rev Neurol 2014;10:554– 569 14. Mesulam M-M, Thompson CK, Weintraub, Ro- galski EJ. Wernicke conundrum and the anat- omy of the comprehension in primary progres- sive aphasia. Brain 2015;138:2423-2437. 15. Gorno-Tempini ML. Classification of primary progressive aphasia and its variants. Neurology 2011;76:1006-1014 16. Marshall CR, Hardy CJD, Volkmer A, Russell LL, Bond RL et al. Primary progressive aphasia: a clinical approach J Neurol 2018;265:1474- 1490 17. Selley WW, Crawford RK, Zhou J, Miller B, Greicius MD. Neurodegenerative diseases tar- get large-scale human brain networks. Neuron 2009; 62:47-52 18. Matias-Guiu JA, Cabrera-Martin MN, Matías- Guiu J, Carrera JL. FDG-PET/CT or MRI for the diagnosis of primary progressive aphasia? Am J Neuroradiol 2017;38 19. Grossman M, Irwin DJ. Primary progres- sive aphasia and stroke aphasia. Continuum 2018;24:745-767 20. Spinelli EG, Mandelli ML, Miller ZA, Santos- Santos MA, Wilson SM y col. Typical and atyp- ical pathology in primary progressive aphasia variants. Ann Neurol 2017;81:430-443 21. Mesulam M-M, Weintraub S, Rogalski EJ, Wieneke C, Geula C y Bigio EH. Asymmetry and heterogeneity of Alzheimer`s and fron- totemporal pathology in primary progressive aphasia. Brain 2014;137:1176-1192 22. Villarejo-Galende A, Amyloid pet in primary progressive aphasia: case series and systematic review of the literature. J Neurol 2017;264:121- 130 23. Santos-Santos MA, Rabinovici GD, Laccarino L, Ayakta N, Tammewar G, Lobach I. Rates of amyloid imaging positivity in patients with primary progressive aphasia. JAMA Neurol 2018;75:342-35 24. Joseph KA, Martin PR, Botha H, Schwarz CG, Duffy JR, Clark HM, Machulda MM y col [18F] AV-1451 Tau-PET and primary progressive aphasia. Ann Neurol 2018;83:599-611 25. Makaretz SJ, Quimby M, Collins J, Makris N, McGinnis S, Schultz A, Vasdev N y col. Flor- taucipir tau PET imaging in semantic variant primary progressive aphasia. J Neurol Neuro- surg Psychiatry 2018;89:1024-1031 26. Lee H, Sea S, Lee S-Y, Jeong HJ, Woo S-H, Lee K-M, Lee Y-B y col. [18F]-THK5351 PET imaging in patients with semantic variant pri- mary progressive aphasia. Alzheimer Dis Assoc Disord 2018; 32:62-69 27. Walker LC, Jucker M. Neurodegenerative dis- eases: expanding the prion concept. Annu Rev Neurosci 2015;38:87-103 28. Goñi F, Martá-Ariza M, Peyser D, Herling K, Wisniewski T. Production of monoclonal an- tibodies to pathologic β-sheet oligomeric con- formers in neurodegenerative diseases. Sci Rep. 2017;7:9881. doi: 10.1038/s41598-017-10393-z. NOTA EN PRENSA Han sido publicados varios artículos relevantes: 29. Ohm DT, Kim G, Gefen T, Rademaker A, Wein- traub S, Bigio EH, Mesulam M-M et al. Pro- minente microglial activarion in cortical white matter is selectively associated with cortical atrophy in primary progressive aphasia. Neu- ropathol App Neurobiol 2019;45:216-229 30. Giannini LAA, Xie SX, McMillan CT, Liang M, Williams A, Jester C, Rascovsky et al. Diver- gent patterns of TDP-43 and tau pathologies in primary progressive apjasia. Ann Neurol 2019;85:630-643 31. Borroni B, Alberici A, Buratti. Review: mo- lecular pathology of frontotemporal lobar degenerations. Neuropathol App Neurobiol 2019;45:41-57 BIBLIOGRAFÍA

RkJQdWJsaXNoZXIy ODI4MTE=