Anales de la RANM

143 A N A L E S R A N M R E V I S T A F U N D A D A E N 1 8 7 9 José Ramón de Berrazueta Fernández An RANM · Año 2019 · número 136 (02) · páginas 131 a 144 ES LA MIOCARDIOPATÍA HIPERTRÓFICA APICAL LA EVOLUCIÓN DE UN MINOCA? crónica y permanente. Nuestro caso presentó derra- me pericárdico en la fase aguda de la enfermedad y tanto el Takotsubo como la MCH apical pueden cur- sar con derrame pericárdico. Aunque no se incluye dentro de las patologías o me- canismos en los que se ha descrito el patrón MI- NOCA, a la vista de lo que hemos discutido, el patrón agudo desencadenante de la MCH apical, como es el caso presentado, puede entenderse como un MINOCA microvascular y la isquemia crónica como INOCA (Ischemia with non obs- tructive coronary arteries). 1. DeWood MA, Spores J, Notske R, et al. Preva- lence of total coronary occlusion during the early hours of transmural myocardial infarction..N Engl J Med. 1980;303 :897-902 . 2. Falk E, Shah PK, Fuster V. Coronary plaque dis- ruption. Circulation. 1995;92:657–671. 3. Rentrop KP, Blanke H, Karsch KR, et al. Acute myocardial infarction: intracoronary application of nitroglycerin and streptokinase. Clin Cardiol. 1979;2:354-363. 4. DeWood MA, M.D., Stifter WF, Simpson CS, et al. Coronary arteriographic findings soon af- ter non-Q-wave myocardial infarction. N Engl J Med 1986; 315:417-423 DOI: 10.1056/NEJM 198608143150703. 5. Anderson JL, Adams CD, Antman EM, et al. ACC/ AHA 2007 Guidelines for the Management of Patients With Unstable Angina/Non–ST-Elevation Myocar- dial Infarction. JACC JACC 2007; 50: :e1–157. 6. Ferreira González I, Permanyer-Miralda G, Ma- rrugat J, et al. en representación de los investigado- res del Estudio MASCARA (Manejo del Sindrome Coronario Agudo. Registro Actualizado). Resulta- dos globales. Rev Esp Cardiol. 2008;61:803-816. 7. Gibbons RJ, Holmes DR, Reeder GS, Bailey KR, Hopfenspirger MR, Gersh, BJ. Immediate an- gioplasty compared with the administration of a thrombolytic agent followed by conservative treatment for myocardial infarction. The Mayo Co- ronary Care Unit and Catheterization Laboratory Groups. N Engl J Med. 1993;328:685-691 8. Ibánez B, James S, Agewall S, Antunes MJ, et al. 2017 ESC Guidelines for the management of acute myocar- dial infarction in patients presenting with ST-segment elevation: The task force for the management of acute myocardial infarction in patients presenting with ST- segment elevation of the European Society of Cardio- logy (ESC) Eur Heart Journal 2018; 39: 119–177 9. Barrabés JA, Bardají A, Jiménez-Candil J, et al. En representación de los investigadores del estu- dio DIOCLES. Pronóstico y manejo del síndrome coronario agudo en España en 2012: estudio DIO- CLES. Rev Esp Cardiol. 2015;68:98–106 10. Thygesen K, Alpert JS, Jaffe AS, Simoons ML, Chaitman BR, White H et al. ESC Committee for Practice Guidelines (CPG). Third universal defi- nition of myocardial infarction. Eur Heart J. 2012; 33:2551-2567. doi: 10.1093/eurheartj /ehs184. 11. Tun A, Khan JA. Myocardial Infarction with Normal Coronary Arteries: The Pathologic and Clinical Perspectives. Angiology 2001; 52: 299-304. 12. Beltrame JF. Assessing patients with myocar- dial infarction and nonobstructed coronary arteries (MINOCA). J Intern Med. 2013;273:182– 185. doi: 10.1111/j.1365-2796.2012.02591.x. 13. Scalone G, Niccoli G, Crea F. Editor’s Choice- Pathophysiology, diagnosis and management of MINOCA: an update. Eur Heart J: Acute Car- diovascular Care 2019; 8: 54–62. https://doi. org/10.1177/ 2048872618782414. 14. Pasupathy S, Air T, Dreyer RP, Tavella R, Beltrame JF. Systematic review of patients presenting with suspected myocardial infarction and nonobstruc- tive coronary arteries. Circulation 2015;131:861- 870. DOI: 10.1161/ CIRCULATIONAHA. 114.011201. 15. Agewall S, Beltrame JF, Reynolds HR, et al.; Wor- king Group on Cardiovascular Pharmacotherapy. ESC working group position paper on myocardial infarction with non-obstructive coronary arteries. Eur Heart J 2017; 38: 143–153. 16. Tanaka A, Imanishi T, Kitabata H, Kubo T, Taka- rada S, Kataiwa, H et al. Distribution and Fre- quency of Thin-Capped Fibroatheromas and Ruptured Plaques in the Entire Culprit Coronary Artery in Patients With Acute Coronary Syndro- me as Determined by Optical Coherence Tomo- graphy. Am J Cardiol 2008;102:975–979. 17. Scalone G, Niccoli G, Crea F. Editor’s Choice- Pathophysiology, diagnosis and management of MINOCA: an update. Eur Heart J: Acute Car- diovascular Care 2019; 8: 54–62. https://doi. org/10.1177/ 2048872618782414. 18. Saw J, Mancini GB, Humphries K, et al. Angio- graphic appearance of spontaneous coronary artery dissection with intramural hematoma proven on intracoronary imaging. Catheter Car- diovasc Interv 2016; 87: E54–E61. 19. Saw J, Aymong E, Mancini GB, et al. Nonatheros- clerotic coronary artery disease in young women. Can J Cardiol 2014; 30: 814–819 20. Di Valentino M, Pedrazzini G , Gallino A .Diseccion coronaria posparto. Rev Esp Cardiol. 2007;60:66. 21. Romero-Rodríguez N, Fernández-Quero M a, Manuel Villa Gil-Ortega M et al. Disección coro- naria espontánea y sus implicaciones pronósticas a largo plazo en una cohorte de 19 casos. Rev Esp Cardiol. 2010;63:1088-1091 . 22. Sabaté M. Descifrando la fisiopatología de la di- sección coronaria espontánea. Rev Esp Cardiol. 2018 DOI:10.1016/j.recesp .2019.02.006. 23. Tweet MS, Hayes SN, Pitta SR, et al. Clinical fea- tures, management, and prognosis of spontaneous coronary artery dissection. Circulation 2012; 126: 579–588. 24. Molina-Povedano MA , Riera-Sagrera M, Ferreruela-Serlavos M, Fiol-Sala M . Paro car- díaco recurrente y vasoespasmo coronario. Med Intensiva. 2011;35 :583-587 25. Kaski JC, Crea F, Meran D, et al. Local coronary supersensitivity to diverse vasoconstrictive stimuli in patients with variant angina. Circulation 1986; 74: 1255–1265. BIBLIOGRAFÍA